Don't show me this again. This is one of over 2, courses on OCW. Find materials for this course in the pages linked along the left. No enrollment or registration. Freely browse and use OCW materials at your own pace.
There's no signup, and no start or end dates. Knowledge is your reward. Use OCW to guide your own life-long learning, or to teach others. We don't offer credit or certification for using OCW.Cell Injury ( Part 1 ) : Definition, Causes, Hypoxia, Different Mechanisms of Cell Injury (HD)
Made for sharing. Download files for later. Send to friends and colleagues. Modify, remix, and reuse just remember to cite OCW as the source. Lecture Notes. Badizadegan PDF - 1. Ingber Epithelial Structure and Function 1 hr. Badizadegan PDF - 2. Colvin PDF - 1. Schoen 8 Introduction to Organ Systems Pathology 1 hr. Hu PDF - 1.
Gimbrone Ischemia and Infarction 1 hr. Rennke 12 Genetic Disorders 1 hr. Badizadegan Genetic Progression in Gastrointestinal Neoplasia 1 hr. Crum 16 Mid-Term Exam 1 hr. Lymphoid Neoplasms 1 hr.
Brugnara 18 Introduction to the Nervous System 1 hr. Toxic Proteins and Neurodegenerative Diseases 1 hr. CNS Neoplasia 1 hr. Louis 19 Diseases of Skeletal Muscle 1 hr.This blog contains a compilation of lecture notes of various medical subjects. Most of them are in. This blog will be helpful for Medical, Dental and Paramedical students in understanding various topics which are prepared by the topmost Doctors in the medical field. This blog will also be helpful for the Practicioners in refreshing there knowledge.
All comments and suggestions should be sent to edoctorforhelp gmail. Post a Comment. Medical Lecture Notes. Medical Android. Search This Blog.
Home Contact me. Vote For This Blog. Click on the images to download presentation. Posted by Dr. Rohini at AM. Labels: Pathology. Newer Post Older Post Home. Subscribe to: Post Comments Atom. Medical Books Online Loading Popular Posts. Pathology Robbins Lecture Notes all Chapters.Rollins, Ph. Thank Prof for your sharing knowledge on Epidemiology. Thanks for sharing your materials with us all. They certainly make teaching epidemiology to undergraduate enjoyable.
These lectures were very useful. I am a PhD student of Epidemiology. I find these PPT to be very helpful in my studies as well as in my future professional careers. Post a Comment. Unable to find out your topic in this website,Then use our special powerpoint search engine below. Use Search Box to find out lecture topics.
Concise Pathology, 3e
If you are going to prepare a seminar you can compare with multiple similar presentations given here, some times if you are busy you can use these slides as such without any modification or with slight modification. Then visit www. Newer Post Older Post Home. Subscribe to: Post Comments Atom.
Chapter One Introduction to Pathology - PowerPoint PPT Presentation
About Me Dr. Join My Facebook Page. My Blogs. Medical Students and Doctors World. Anatomy Android Mobile Application for medical students. Read the comments!!!! My Google Plus Profile Google. Useful External Websites StudyingMed. We do not host or modify the presentations. These lecture notes are sole property of original uploaders. We are just sharing them for helping medical education world wide. If you find any copyrighted slides inform me i will take necessary actions. If any of of you have a good personal power point presentation Email me i will upload it here.
Robbins Pathology PowerPoint PPT Presentations
The first chapters of the text discuss general concepts and diseases affecting the body as a whole. Later chapters consider the various organ systems and their diseases.
Pathology, Lecture 1 Introduction to Pathology (Slides)
The Tenth Edition boasts a wealth of new disease photos, new and expanded case studies, and a robust student and instructor ancillary package. Each new print copy includes Navigate 2 Advantage Access that unlocks a comprehensive and interactive eBook with animations, student practice activities and assessments, a full suite of instructor resources, and learning analytics reporting tools.
Written for the following course which are often offered through the Nursing or Health Professional Department:. Newly Acquired. Available Resources.Toggle navigation. Help Preferences Sign up Log in.
Featured Presentations. Recommended textbook: Vinay Kumar, Abul K. General Objectives What is the responsibility of. Students often ask me this question in the Il y a actuellement 4 classes de ch mokines: C-X-C ou ch mokines alpha, dont les Anatomical Pathology RPadmore ottawahospital. Cardiovascular Pathology modification of Dr. Pathology - Cytopathology. Autopsy Pathology. Clinical Pathology CLS Applies scientific laboratory methods relevant to patient care, health promotion, Secondary Biliary Cirrhosis.
Primary Biliary Cirrhosis. Big 4. Big 3. Pathology of the Pancreas - Pathology of the Pancreas. Alyssa Krasinskas, MD.
January 6, Fat necrosis and saponification release of lipolytic enzymes Pathology of the Pancreas. Pathologie hpatobiliaire et pancratique - Analyse d'une histoire clinique: facteurs suggestifs d'une h patopathie, diagnostic Illustrer les pathologies h patiques, biliaires et pancr atiques les Analyse d'une histoire clinique: facteurs suggestifs d'une h patopathie, diagnostic Pathology of the Small Bowel - May be related to enterotoxigenic organisms E. May be related to enterotoxigenic organisms E.
Perinatal and pediatric pathology - Perinatal and pediatric pathology Retinoblastoma Wilms tumor Fetal atelectasis 24 Hyaline membrane disease 76 Medulloblastoma 83 Craniopharyngioma Perinatal and pediatric pathology Retinoblastoma Wilms tumor Fetal atelectasis 24 Hyaline membrane disease 76 Medulloblastoma 83 Craniopharyngioma General Pathology Illustrations Dr.
Cooperstein - In general, foods that cause gout are high in fat, which is why dietary In general, foods that cause gout are high in fat, which is why dietary Hypopituitarism: Clinical Evaluation Hypopituitarism: Pituitary Apoplexy Hypopituitarism: ischemic necrosis of anterior lobe Sheehan syndrome Byron E.
Crawford, MD Board Exams will be on the designated Friday Make-up Test.Copy embed code:. Automatically changes to Flash or non-Flash embed. WordPress Embed Customize Embed. URL: Copy. Presentation Description No description available. Explain the role of pathophysiology in the diagnosis and treatment of disease. Use appropriate terminology. Explain the importance of a patient's medical history.
Describe common cellular adaptations and possible reasons for the occurrence of each. Identify precancerous cellular changes. List the common causes of cell damage. Describe the common types of cell necrosis and possible outcomes.
Chapter 1. Introduction to Pathophysiology: Introduction to Cellular Changes PowerPoint Presentation: Homeostasis : Cells tend to preserve their immediate environment and intracellular environment. Cell injury could be reversible e. Pathophysiology involves the study of functional or physiologic changes in the body that result from disease processes.
This subject builds on knowledge of the normal structure, and function of the human body.
As such, pathophysiology includes some aspects of pathology histopathologythe laboratory study of cell and tissue changes associated with disease. When a sufficient number of cells is involved, the entire tissue or organ diminishes in size.
Cells are not dead. Causes of atrophy include: 1- Decreased workload e. Hypertrophy can be physiologic or pathologic and is caused either by increased functional demand or by specific hormonal stimulation.
Physiologic hyperplasia is divided into 1 hormonal hyperplasia, 2 compensatory hyperplasia, Most forms of pathologic hyperplasia are instances of excessive hormonal or growth factor stimulation.
Hyperplasia could be precancerous. Is a reversible change in which one differentiated adult cell type is replaced by another differentiated adult cell type. It might be protective adaptive mechanism e. Thyroid-diffuse hyperplasia Graves disease Cell Damage and Necrosis: Cell Damage and Necrosis There are many ways of injuring cells in the body, including: ischemiaor deficit of oxygen in the cells, due to respiratory problems or circulatory obstruction; physical agents, excessive heat or cold, or radiation exposure; mechanical damage such as pressure or tearing of tissue; chemical toxins or foreign substances; microorganisms such as bacteria, viruses, and parasites; abnormal metabolites accumulating in cells; nutritional deficits; and imbalance of fluids or electrolytes.
Cell lysis releases destructive lysosomal enzymes into the tissue, which cause inflammation swelling, redness and pain as well as damage to nearby cells. The enzymes released from the dead cells can diffuse into the blood, providing helpful clues in blood tests that indicate the type of cells damaged. It is characterized by cell swelling, denaturation of cytoplasmic proteins, and enzymatic digestion of the cell. Implies preservation of the basic structural outline of the cell or tissue for a span of days.Cell Injury - Suggested readings from Robbins 10th ed.
Clinical History: A year-old man with history of uncontrolled diabetes and hypertension presented to the emergency department with a 3-day history of nagging chest pain. Four hours earlier, the chest pain got much worse and radiated to both arms and his neck. It was accompanied by diaphoresis and dyspnea.
Heart rate is regular and there is trace bilateral edema. The cardiac catherization lab treats him with thrombolytic agents for a diagnosis of ST elevation myocardial infarction.
Approximately 20 minutes after infusion begins, he experiences ventricular tachycardia and dies. Normal heart tissue sections demonstrate no evidence of fibrosis or hemorrhage.
Cardiac myocytes have moderately sized centrally located nuclei. Normal myocytes are not brightly eosinphilic. Normally no inflammation is seen. Normal cardiac myocytes do not show hypertrophy. Increased eosinophilia of the dead myocytes is most directly linked to which of the following molecular events?
Which of the following is the most likely explanation for the onset of ventricular tachycardia following initiation of thrombolytic therapy? Clinical History: A year-old woman with a long history of hypertension and a remote myocardial infarction presented to the emergency department with a two-hour history of left-sided weakness. While in the emergency department, she developed left-sided hemiparesis. She was admitted to the hospital for observation; however, her condition worsened and she went into a coma.
She died 17 days after admission. The normal gastric mucosa of the fundus contains superficial fovea arranged in leaf like fronds and deeper gastric glands. The foveolar cells secrete mucin. The gastric glands include mucous cells, parietal cells, chief cells and enteroendocrine cells. Which of the following processes did the necrotic cells in this specimen undergo?
Which of the following is a consequence of this process? In this ischemic environment, the mitochondria are unable to maintain normal oxidative phosphorylation, resulting in release of reactive oxygen species into the cytoplasm.
Which of the following is the most direct consequence of this release?