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Chapter one introduction to pathology ppt

By | 13.10.2020

chapter one introduction to pathology ppt

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Cell Injury ( Part 1 ) : Definition, Causes, Hypoxia, Different Mechanisms of Cell Injury (HD)

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chapter one introduction to pathology ppt

Home Contact me. Vote For This Blog. Click on the images to download presentation. Posted by Dr. Rohini at AM. Labels: Pathology. Newer Post Older Post Home. Subscribe to: Post Comments Atom. Medical Books Online Loading Popular Posts. Pathology Robbins Lecture Notes all Chapters.Rollins, Ph. Thank Prof for your sharing knowledge on Epidemiology. Thanks for sharing your materials with us all. They certainly make teaching epidemiology to undergraduate enjoyable.

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Concise Pathology, 3e

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Chapter One Introduction to Pathology - PowerPoint PPT Presentation

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Robbins Pathology PowerPoint PPT Presentations

By using the site, you consent to the placement of these cookies. Read our Cookie Policy to learn more. The product you are seeking was not found, or has been deleted. Please go back and try again. Updated and reorganized to provide a more accessible, student-friendly experience, Crowley's An Introduction to Human Disease, Tenth Edition provides readers with a clear, well-illustrated explanation of the structura. Updated and reorganized to provide a more accessible, student-friendly experience, Crowley's An Introduction to Human Disease, Tenth Edition provides readers with a clear, well-illustrated explanation of the structural and functional changes associated with disease, the clinical manifestations of disease, and how to determine treatment.

The first chapters of the text discuss general concepts and diseases affecting the body as a whole. Later chapters consider the various organ systems and their diseases.

Pathology, Lecture 1 Introduction to Pathology (Slides)

The Tenth Edition boasts a wealth of new disease photos, new and expanded case studies, and a robust student and instructor ancillary package. Each new print copy includes Navigate 2 Advantage Access that unlocks a comprehensive and interactive eBook with animations, student practice activities and assessments, a full suite of instructor resources, and learning analytics reporting tools.

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chapter one introduction to pathology ppt

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Cooperstein - In general, foods that cause gout are high in fat, which is why dietary In general, foods that cause gout are high in fat, which is why dietary Hypopituitarism: Clinical Evaluation Hypopituitarism: Pituitary Apoplexy Hypopituitarism: ischemic necrosis of anterior lobe Sheehan syndrome Byron E.

Crawford, MD Board Exams will be on the designated Friday Make-up Test.Copy embed code:. Automatically changes to Flash or non-Flash embed. WordPress Embed Customize Embed. URL: Copy. Presentation Description No description available. Explain the role of pathophysiology in the diagnosis and treatment of disease. Use appropriate terminology. Explain the importance of a patient's medical history.

Describe common cellular adaptations and possible reasons for the occurrence of each. Identify precancerous cellular changes. List the common causes of cell damage. Describe the common types of cell necrosis and possible outcomes.

Chapter 1. Introduction to Pathophysiology: Introduction to Cellular Changes PowerPoint Presentation: Homeostasis : Cells tend to preserve their immediate environment and intracellular environment. Cell injury could be reversible e. Pathophysiology involves the study of functional or physiologic changes in the body that result from disease processes.

This subject builds on knowledge of the normal structure, and function of the human body.

chapter one introduction to pathology ppt

As such, pathophysiology includes some aspects of pathology histopathologythe laboratory study of cell and tissue changes associated with disease. When a sufficient number of cells is involved, the entire tissue or organ diminishes in size.

Cells are not dead. Causes of atrophy include: 1- Decreased workload e. Hypertrophy can be physiologic or pathologic and is caused either by increased functional demand or by specific hormonal stimulation.

Physiologic hyperplasia is divided into 1 hormonal hyperplasia, 2 compensatory hyperplasia, Most forms of pathologic hyperplasia are instances of excessive hormonal or growth factor stimulation.

Hyperplasia could be precancerous. Is a reversible change in which one differentiated adult cell type is replaced by another differentiated adult cell type. It might be protective adaptive mechanism e. Thyroid-diffuse hyperplasia Graves disease Cell Damage and Necrosis: Cell Damage and Necrosis There are many ways of injuring cells in the body, including: ischemiaor deficit of oxygen in the cells, due to respiratory problems or circulatory obstruction; physical agents, excessive heat or cold, or radiation exposure; mechanical damage such as pressure or tearing of tissue; chemical toxins or foreign substances; microorganisms such as bacteria, viruses, and parasites; abnormal metabolites accumulating in cells; nutritional deficits; and imbalance of fluids or electrolytes.

Cell lysis releases destructive lysosomal enzymes into the tissue, which cause inflammation swelling, redness and pain as well as damage to nearby cells. The enzymes released from the dead cells can diffuse into the blood, providing helpful clues in blood tests that indicate the type of cells damaged. It is characterized by cell swelling, denaturation of cytoplasmic proteins, and enzymatic digestion of the cell. Implies preservation of the basic structural outline of the cell or tissue for a span of days.Cell Injury - Suggested readings from Robbins 10th ed.

Clinical History: A year-old man with history of uncontrolled diabetes and hypertension presented to the emergency department with a 3-day history of nagging chest pain. Four hours earlier, the chest pain got much worse and radiated to both arms and his neck. It was accompanied by diaphoresis and dyspnea.

Heart rate is regular and there is trace bilateral edema. The cardiac catherization lab treats him with thrombolytic agents for a diagnosis of ST elevation myocardial infarction.

Approximately 20 minutes after infusion begins, he experiences ventricular tachycardia and dies. Normal heart tissue sections demonstrate no evidence of fibrosis or hemorrhage.

Cardiac myocytes have moderately sized centrally located nuclei. Normal myocytes are not brightly eosinphilic. Normally no inflammation is seen. Normal cardiac myocytes do not show hypertrophy. Increased eosinophilia of the dead myocytes is most directly linked to which of the following molecular events?

Which of the following is the most likely explanation for the onset of ventricular tachycardia following initiation of thrombolytic therapy? Clinical History: A year-old woman with a long history of hypertension and a remote myocardial infarction presented to the emergency department with a two-hour history of left-sided weakness. While in the emergency department, she developed left-sided hemiparesis. She was admitted to the hospital for observation; however, her condition worsened and she went into a coma.

She died 17 days after admission. The normal gastric mucosa of the fundus contains superficial fovea arranged in leaf like fronds and deeper gastric glands. The foveolar cells secrete mucin. The gastric glands include mucous cells, parietal cells, chief cells and enteroendocrine cells. Which of the following processes did the necrotic cells in this specimen undergo?

Which of the following is a consequence of this process? In this ischemic environment, the mitochondria are unable to maintain normal oxidative phosphorylation, resulting in release of reactive oxygen species into the cytoplasm.

Which of the following is the most direct consequence of this release?


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